https://ogma.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:28034 Wed 11 Apr 2018 16:26:21 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:15919 Wed 11 Apr 2018 10:53:24 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:33984 Wed 04 Sep 2019 09:39:10 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:17330 Sat 24 Mar 2018 08:01:51 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:27038 h currents, and islet cell morphology. Although both Typical and Atypical CR-expressing neurons responded to noxious peripheral stimulation, the excitatory drive onto Typical CR-expressing neurons was much stronger. Furthermore, Atypical CR-expressing cells comprise at least two functionally distinct subpopulations based on their responsiveness to noxious peripheral stimulation and neurochemical profile. Together our data suggest CR expression is not restricted to excitatory neurons in the SDH. Under normal conditions, the contribution of 'Typical' excitatory CR-expressing neurons to overall SDH excitability may be limited by the presence of A-type potassium currents, which limit the effectiveness of their strong excitatory input. Their contribution may, however, be increased in pathological situations where A-type potassium currents are decreased. By contrast, 'Atypical' inhibitory neurons with their excitable phenotype but weak excitatory input may be more easily recruited during increased peripheral stimulation.]]> Sat 24 Mar 2018 07:25:24 AEDT ]]>